"A Twist of Fate: Torsades de Pointes Following Left Insular Stroke" by Hazem Alakhras, Michelle Darmadi et al.
 

A Twist of Fate: Torsades de Pointes Following Left Insular Stroke

Document Type

Conference Proceeding

Publication Date

4-1-2025

Publication Title

Journal of the American College of Cardiology

Abstract

Background: Torsades de Pointes (TdP) is a life-threatening form of ventricular tachycardia that occurs in patients with a prolonged QT interval. While acute strokes are known to cause various EKG abnormalities, including QT prolongation, ST-segment changes, and T-wave inversions, TdP is a rarely reported complication, with only two documented cases in the literature. Case: A 75-year-old female with no prior cardiac history presented with expressive aphasia, prompting a stroke workup. Brain MRI confirmed an acute ischemic stroke of the left insular cortex; however, thrombolytics were withheld due to a low NIHSS score. Initially, she had a normal sinus rhythm with a QTc of 438 ms. However, the next day, she experienced dizziness and telemetry revealed TdP. A 12-lead EKG showed a significantly prolonged QTc of 685 ms, which had not been present earlier. Decision-making: She was treated with IV magnesium, and started on isoproterenol and lidocaine drips. Upon further monitoring, her QTc normalized to 460 ms, the drips were gradually weaned off, and she was eventually discharged with mexiletine. Of note, her echocardiogram on admission showed a reduced ejection fraction of 25%, but it improved to 50% on discharge. Cardiac catheterization revealed non-obstructive coronary artery disease, ruling out ischemic heart disease. Other common causes of QT prolongation, such as electrolyte imbalances, drug reactions, and thyroid abnormalities were also ruled out. We suspect that the arrhythmia was neurogenic as it resolved after the ischemic event passed and due to the location of the infarct. The insular cortex is known to regulate cardiovascular function, with the right side increasing sympathetic tone and the left side enhancing parasympathetic tone. In this case, unopposed right insular activity likely triggered the arrhythmia. Conclusion: To our knowledge, this is the first reported case of TdP resulting from a left insular cortex infarction. This case underscores the importance of telemetry following an acute stroke and highlights the need for further research to identify which brain cortices are at higher risk of developing life-threatening arrhythmias.

Volume

85

Issue

12 Suppl

First Page

3671

Comments

American College of Cardiology Meeting, March 29-31, 2025, Chicago, IL

Last Page

3671

DOI

10.1016/S0735-1097(25)04155-5

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