"Metabolomic Insights Into Fibrotic Lung Disease Differentiation From C" by Faizan Faizee, Zaid Alnabulsi et al.
 

Metabolomic Insights Into Fibrotic Lung Disease Differentiation From Chronic Obstructive Pulmonary Disease

Document Type

Conference Proceeding

Publication Date

2025

Publication Title

American Journal of Respiratory and Critical Care Medicine

Abstract

Background: Fibrotic lung disease encompasses idiopathic pulmonary fibrosis (IPF) and other interstitial lung diseases (ILDs). We investigated distinct metabolic patterns between IPF/ILD and chronic obstructive pulmonary disease (COPD) to enhance our understanding of the underlying disease mechanisms related to disease progression.

Methods: This single-center prospective study enrolled (n=71) participants between December 2021 and October 2022. Participants were categorized as progressive or stable IPF/ILD based on the 2018 IPF guidelines. Metabolite quantification was performed using the biocrates Quant 500 XL assay, a liquid chromatography-mass spectrometry (LC-MS platform), and nuclear magnetic resonance spectroscopy (H NMR). Further, pathway enrichment analysis identified significantly disrupted biochemical pathways.

Results: Of the 715 metabolites accurately quantified, 59 showed significant differences between the combined IPF/ILD groups and COPD controls. The most distinctive metabolic signatures differentiating fibrotic lung disease from COPD were found in glycerolipids class (GL), particularly various triacylglycerol (TG) and diacylglycerol (DG) species (false discovery rate [FDR] q < 0.05). Enrichment analysis comparing IPF/ILD versus COPD revealed significant disruptions in lipid metabolic pathways, with notable alterations in glycerophospholipids and sphingolipids metabolism (FDR q-value < 0.05). These findings demonstrate clear metabolomic distinctions between fibrotic lung diseases and COPD, highlighting specific lipid metabolism pathways as key differentiators.

Conclusion: This study reveals distinct metabolomic signatures that effectively differentiate between ILD and COPD, particularly in lipid metabolism pathways. The identification of specific glycerolipid alterations provides mechanistic insights that might inform future biomarker development, potentially enabling early identification of patients at elevated risk for fibrotic lung disease progression. Further investigation of these metabolic pathways could lead to improved diagnostic tools and targeted therapeutic strategies.

Volume

211

First Page

A6147

Comments

The American Thoracic Society (ATS) International Conference, May 16-21, 2025, San Francisco, CA.

DOI

10.1164/ajrccm.2025.211.Abstracts.A6147

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